The prevalence of age-related neurodegenerative diseases is rapidly increasing whilst effective treatment and prevention strategies still need to be established. The urgency to discover risk-associated and causative molecular mechanisms to improve our understanding of the pathogenesis of these disorders is therefore high.

Accumulating evidence including recent cutting-edge discoveries propose both disease-specific and disease-overlapping roles of glial cells in the pathophysiology of neurodegenerative diseases. Furthermore, regulation and function of the cerebrovasculature, blood-flow and the integrity of the blood-brain-barrier are frequently affected in neurodegenerative conditions.

The 6th International Conference on Molecular Neurodegeneration (ICMN2020) will offer a series of invited talks by well-renowned speakers to discuss Glial and Vascular Contributions to Neurodegenerative Diseases. The combination of invited talks with abstract-selected short talks and poster presentations will provide excellent opportunities for productive discussions, networking, and collaboration to further our understanding on the genetic, cell and molecular processes responsible for the most common neurodegenerative disorders.

Save the date and join us at ICMN2020 in Bangkok, Thailand, May 21-23 2020 in Bangkok, Thailand!

 

ICMN2020 Organizing Committee
(Guojun Bu, Henrietta Nielsen, Robert Vassar, Huaxi Xu, Hui Zheng)

ICMN2020 THEME

Glial and Vascular Contributions to Neurodegenerative Diseases

Neurodegenerative diseases collectively refer to neurological disorders that result from neurodegeneration and include, but are not limited to, Alzheimer’s disease, Parkinson’s disease, Huntington’s disease, and prion diseases. These diseases, which are often associated with advanced aging and display varying degrees of dementia, have become a significant public health issue as humans live longer and the aging population grows larger. Recent advances in the molecular and cellular mechanisms underlying the pathogenesis of these neurodegenerative disorders have allowed for a better understanding of the disease mechanisms.

The conference is organized by Molecular Neurodegeneration, the leading peer-reviewed open access journal in the neuroscience category and the official journal of the BrightFocus Foundation.

MOLECULAR NEURODEGENERATION

The conference is organized by Molecular Neurodegeneration, the leading peer-reviewed open access journal in the neuroscience category and the official journal of the BrightFocus Foundation.

Molecular Neurodegeneration is an open access, peer-reviewed journal that encompasses all aspects of neurodegeneration research at the molecular and cellular levels.

Open access
All articles published by Molecular Neurodegeneration are made freely and permanently accessible online immediately upon publication, without subscription charges or registration barriers. Further information about open access can be found here.  Authors of articles published in Molecular Neurodegeneration are the copyright holders of those articles and have granted to any third party, in advance and in perpetuity, the right to use, reproduce or disseminate your article, according to the BioMed Central license agreement.  For those who are US government employees or are prevented from being copyright holders for similar reasons, BioMed Central can accommodate non-standard copyright lines. 

ORGANIZING COMMITTEE

Guojun Bu (Molecular Neurodegeneration co-editor in chief) 
Mayo Clinic FL, USA
 
Henrietta Nielsen (Molecular Neurodegeneration senior editor)
Stockholm University Sweden, Roskilde University Denmark
 
Robert Vassar (Molecular Neurodegeneration associate editor)
Northwestern University Medical School, USA.
 
Huaxi Xu (Molecular Neurodegeneration co-editor in chief)
Sanford Burnham Prebys Medical Discovery Institute, USA
 
Hui Zheng (Molecular Neurodegeneration associate editor)
Baylor College of Medicine, USA

Review Organizing Committee bios here.

SESSIONS

The event will consist of guest podium presentations, panel discussions and selected poster presentations.  

The abstract submission platform will open later this winter.  

AGENDA - DAY 1

THURSDAY, May 21
07:30 REGISTRATION
08:30 WELCOME REMARKS
08:45 SESSION I: Glial Contributions to Neurodegeneration
10:25 COFFEE BREAK/EXHIBITS
10:45 SESSION II: Genomic and Functional Studies of Glia
12:25 LUNCH/EXHIBITS
13:45 SESSION III: Novel Systems and Approaches
15:25 BREAK/EXHIBITS
15:45 SHORT TALKS I
17:15 PANEL DISCUSSION
16:35 SHORT TALKS II
18:15 POSTERS/RECEPTION

AGENDA - DAY 2

FRIDAY, May 22
07:30 REGISTRATION
08:30 SESSION IV: ApoE and Vascular Contributions to Neurodegeneration
10:10 BREAK/EXHIBITS
10:30 SESSION V: Pathogenic mechanisms of Neurodegeneration (AD and ADRD)
12:35 LUNCH/EXHIBITS
14:00 SESSION VI: Pathogenic Mechanisms of Neurodegeneration (PD and ALS)
15:40 BREAK/EXHIBITS
16:00 SHORT TALKS
17:30 PANEL DISCUSSION
18:30 POSTER SESSION

AGENDA - DAY 3

SATURDAY, May 23
07:30 Registration
08:30 SESSION VII: Neuropathology and Biomarkers
10:10 BREAK/EXHIBITS
10:30 SESSION VIII: Therapeutic Insights
11:45 AWARDS
12:00 CLOSING
12:05 LUNCH and NETWORKING

COUNTDOWN TO ABSTRACT OPENING

Day(s)

:

Hour(s)

:

Minute(s)

:

Second(s)

COUNTDOWN TO CONFERENCE

Day(s)

:

Hour(s)

:

Minute(s)

:

Second(s)

IMPORTANT DEADLINES

Abstract submission opens December 9, 2019
Abstract submission closes January 27, 2020
Notification of abstract acceptance on or around February 15, 2020
Early-bird registration ends February 28, 2020

2018 GALLERY